GLOSSARY

Parathyroid Gland produces PTH and differs from the thyroid gland.

Intestine, kidney and bone jointly regulate calcium and phosphate, with related but distinct roles for PTH, active vitamin D and FGF23. Normal blood concentration does not always prove adequate intake or complete skeletal health because stores are also regulated. One postexercise hormone change cannot establish mineral deficiency. Dietary assessment, vitamin status and clinical context come before conclusions about supplement doses rather than treating every endocrine change as a nutrient shortage.

Hormones are chemical signals whose effects depend on target cells and appropriate receptors. Endocrine generally describes communication through blood, while local and neural signals also contribute. One substance can have several roles and sources. Calling something a metabolic or appetite hormone does not imply an exclusive effect; interpretation requires the signaling network, food and activity rather than assigning the body's response to one molecule alone.

Synthesis, storage, secretion, transport and clearance are distinct processes. Blood concentration reflects entry and removal rather than directly measuring secretion; exercise-related plasma-volume changes can also affect it. Half-life depends on hormone form and method and is not identical to the duration of all effects. Comparisons therefore need sampling time, previous food and recent activity standardized or recorded rather than interpreting isolated concentration differences without context.

Hormonal axes use feedback and often pulsatile and time-dependent patterns. A rise can be compensatory and a decline temporarily adaptive, neither diagnosing disease without context. Hormone ratios depend on numerator, denominator, timing and units. Sports nutrition does not aim to maximize every hormone; the practical goal is adequate supply and appropriate physiological responses, evaluated with health and performance rather than an isolated ratio or peak.

Low energy availability means insufficient energy remains after exercise for required bodily functions; it is not synonymous with low carbohydrate or weight loss alone. Problematic exposure can affect reproductive, thyroid, bone and appetite systems with variable patterns. Restoring supply and reviewing training load are central to assessment. One normal hormone or a universal numerical cutoff cannot confirm or exclude every case, so symptoms, history and clinical evaluation must be integrated.

Hormonal imbalance is a broad description rather than a standalone diagnosis from an online symptom list. Deficiency, excess and resistance differ, and reference ranges depend on method, age and conditions. Medicines, illness, sleep and energy status also matter. Persistent symptoms need appropriate assessment to identify causes; choosing supplements or manipulating hormones from one isolated test cannot replace evidence-based evaluation or establish a universal athletic hormone target.