GLOSSARY

Omentin is an intelectin-related protein studied in visceral adipose biology.

Adipose tissue contains several cell types, so adipokines are not restricted to adipocyte secretion. Molecules can associate with inflammation or insulin sensitivity without proving causation or treatment utility. Human and animal sources may differ. Adipokine ratios are not universal athletic targets; assays, units and energy state must be specified. A research pathway cannot alone justify a supplement or establish that changing one circulating protein will improve performance.

Muscle, liver, bone and adipose tissue produce signals alongside their other functions. Myokine, hepatokine and lipokine group molecules by source or nature, with unequal evidence across members. Blood detection does not prove a unique source. Cellular and animal findings about muscle or bone cannot be converted directly into human hormonal treatment or performance products; human outcomes and measurement methods must be established rather than assuming all organ-derived signals have proven athletic uses.

Leptin mainly conveys adipose information about energy stores and availability and also responds to intake changes. Its relation to fat mass is not perfectly fixed; reduced intake can affect it before major weight change. High leptin does not necessarily mean greater satiety, nor is low leptin always a separate disease. Leptin resistance is a regulatory concept not conclusively diagnosed from hunger or one concentration alone without clinical context.