GLOSSARY

Lipolysis hydrolyzes triglycerides to release fatty-acid and glycerol components.

Lipolysis releases fatty acids and glycerol from triglycerides in adipose or intramuscular stores. Release differs from oxidation; some fatty acids are re-esterified. Greater mobilization or blood levels therefore do not prove net body-fat loss. Hormones, blood flow and tissue demand influence handling. Longer-term balance matters for body composition rather than assuming a lipolysis marker establishes that an exercise session or supplement burns more stored fat overall.

Lipid stores and turnover reflect entry, synthesis, breakdown and oxidation across tissues. Packaging, transport and re-esterification can accompany release. Mobilization, burning and net body-fat loss are therefore different reports. Athletes should assess body-composition trends alongside intake, performance and recovery. Maximizing momentary fat oxidation may overlook session fueling, and no isolated blood fatty-acid measure or pathway rate replaces a longer-term account of energy intake and use.

Esterification attaches fatty acids to suitable molecules such as glycerol structures; re-esterification reuses released fatty acids. Triglyceride assembly differs from making new fatty-acid chains. Storage is normal physiology, not automatically damage. Body-composition goals require time-integrated synthesis, breakdown and intake. Observing a post-meal storage step does not determine the result over weeks or justify avoiding all foods that transiently promote storage during ordinary recovery and feeding.